Classification & Workup
Group 1 through 5 — a common phenotype, five very different diseases.
Pulmonary Vascular Remodeling in PAH
Group 1 PAH arises from progressive remodeling of the distal pulmonary arterioles driven by endothelial dysfunction, with reduced production of vasodilators (nitric oxide, prostacyclin) and relative excess of vasoconstrictors (endothelin-1). This imbalance promotes smooth muscle and endothelial cell proliferation, in situ microthrombosis, and formation of plexiform lesions that progressively obliterate the vascular lumen. The result is a fixed rise in pulmonary vascular resistance that increases right ventricular afterload, distinguishing PAH from pulmonary venous hypertension due to left heart disease, where the elevated pressure is transmitted backward rather than generated by primary arteriolar disease. Unchecked, this remodeling drives right ventricular hypertrophy, dilation, and eventual right heart failure.
Hemodynamic Definition (2022 ESC)
WHO Groups
Workup
TTE (screen). CT chest/PE. PFTs. V/Q scan (mandatory to exclude CTEPH). Sleep study. HIV, autoimmune workup. RHC confirms diagnosis and enables vasoreactivity testing.