Atrial Septal Defect
The commonly missed adult congenital lesion.
Shunt Physiology
An unrepaired ASD allows left-to-right shunting because the right ventricle is more compliant than the left, not because of a pressure gradient; shunt magnitude therefore depends on defect size and the relative compliance of the two ventricles. Chronic left-to-right flow causes RV and pulmonary artery volume overload with progressive RV dilation, while the pulmonary vasculature is gradually exposed to increased flow. Over decades this can produce pulmonary vascular remodeling and rising pulmonary vascular resistance; if resistance exceeds systemic resistance, shunt flow reverses right-to-left, producing Eisenmenger syndrome with cyanosis. This natural history is the rationale for closing hemodynamically significant defects before irreversible pulmonary vascular disease develops.
Types
Diagnosis
Widely fixed split S2, pulmonary flow murmur. Echo with agitated saline (bubble study) confirms shunt. TEE preferred for procedural planning.